Covid-19 vaccine induced myocarditis may be different and perhaps more dangerous than “regular” myrocarditis. It may also have no externally manifested symptoms. From Madhava Setty, M.D. at childrenshealthdefense.org:
Autopsies of two teenage boys who died days after receiving Pfizer’s COVID vaccine prove the vaccine caused their deaths. Pathological findings suggest there may be a way to distinguish SARS-COV-2 infection-induced myo/pericarditis from vaccine-induced cardiac injury. Vaccine-induced heart injury can be sub-clinical, but how often?
Pathologists who examined the autopsies of two teenage boys who died days after receiving Pfizer’s COVID-19 vaccine concluded the vaccine caused the teens’ deaths.
The three pathologists, two of whom are medical examiners, published their findings Feb. 14 in an early online release article, “Autopsy Histopathologic Cardiac Findings in Two Adolescents Following the Second COVID-19 Vaccine Dose,” in the Archives of Pathology and Laboratory Medicine.
The authors’ findings were conclusive. Two teenage boys were pronounced dead in their homes three and four days after receiving the second Pfizer-BioNTech COVID-19 dose.
There was no evidence of active or previous COVID-19 infection. The teens had negative toxicology screens (i.e. no drugs or poisons were present in their bodies).
These boys died from the vaccine.
Histopathological examination of their cardiac tissue revealed an important new finding: Neither heart demonstrated evidence of typical myocarditis.
Instead, the authors found evidence of microscopic changes consistent with a different form of heart injury called toxic cardiomyopathy. They wrote:
“The myocardial injury seen in these post-vaccine hearts is different from typical myocarditis and has an appearance most closely resembling a catecholamine-mediated stress (toxic) cardiomyopathy.”
The authors further explained what they observed under the microscope:
“Their histopathology does not demonstrate a typical myocarditis … In these two post-vaccination instances, there are areas of contraction bands and hypereosinophilic myocytes distinct from the inflammation.
“This injury pattern is instead similar to what is seen in the myocardium of patients who are clinically diagnosed with Takotsubo, toxic or ‘stress’ cardiomyopathy, which is a temporary myocardial injury that can develop in patients with extreme physical, chemical, or sometimes emotional stressors.
“Stress cardiomyopathy is a catecholamine-mediated ischemic process seen in high catecholamine states in the absence of coronary artery disease or spasm. It has also been called ‘neurogenic myocardial injury’ and ‘broken heart syndrome.’”
The pathologists determined there was a different mechanism of heart injury at play in these two boys, distinct from a purely infectious process that would result directly from a viral infection like COVID-19.
